Longevity Centres IV Lipid Exchange

Lipid Infusion Therapy

Indications: Chronic Metabolic Oxidative State, Cholesterol Abnormalities

Contains: Phosphatidyl Choline, Glutathione



 

The Longevity Centres of America offers IV Phosphatidyl Choline + IV Glutathione infusions as an effective Anti-Aging Modality. Lipid therapy offers a 1-2 punch against: Cholesterol Irregularities, Cellular Oxidation, Oxygen Radicals, Liver Toxicity, Low Immunity and Metabolic Pollutants.

Glutathione
Glutathione is known to medicine since 1888. In the last twenty years, this protein has been the object of intense research. Now, more than 57,000 scientific articles on GSH are published on MEDLINE and this library is enriched by 3,000 to 5,000 new articles each year.

Glutathione plays three vital roles in the body:

  • maintains the immune system active

  • powerful antioxidant and a free radicals scavenger

  • neutralizes pollutants and detoxifies the cells.

Glutathione is synthesized by using three amino acids: glycine, glutamic acid and cysteine. In our food, we find a sufficient quantity of glycine and glutamic acid. However, cysteine is a problem and a limiting factor in the glutathione synthesis. The reason is simple, food cooking and pasteurization of milk products almost destroy cysteine completely. The consequence of this situation is that with age, we lose up to 40% of our reserves and this makes us vulnerable to the development of disease. Here is what Dr. Jimmy Gutman, Emergency Physician and expert speaker on Glutathione, says: "In fact, your life depends on glutathione. Without it, your cells would disintegrate from unrestrained oxidation, your body would have little resistance to bacteria, viruses and cancer, and your liver would shrivel up from the eventual accumulation of toxins".

Doctors and experts agree that such a discovery happens once per generation and in a few years, Glutathione will be as part of standard tests as blood and cholesterol tests are today.

The health of the membrane is synonymous with the health of the entire organism. Toxins have an affinity for fatty acids; they literally take up residence in the lipid environment and in so doing, weaken and disrupt. The probable result is early apoptosis, premature death of the cell. Generally, normal mitosis provides for new cellular growth to maintain the health of the body, i.e. the previous discussion on photo receptors. However, toxicity's affinity for lipids can easily redistribute toxins and diseased toxic lipids into the new growth. In a healthy state with adequate glutathione and ascorbate to bind the toxins before they take up new residence, the body can keep the bad guys under control. However, if defenses are weak, toxins can continually be redistributed and eventually hide in the CNS and bone where the regeneration process is at a slower pace. The goal of detoxification is to encourage regrowth with a renewed effort at the correct balancing of the essential nutrients, with the exchange of high energy lipids (PUPA and HUP A) to fuel regeneration and the eventual detoxification process.

Phosphatidyl Choline
Detoxification of neurotoxins requires that the cell membrane is nourished with balanced essential fatty acids (4:1, plus HUFAs) and supportive phospholipids. Phosphatidylcholine (PC) is the most abundant phospholipid of the cell membrane and protects the liver, with its 33,000 square meters of membrane, against toxicity and infection. The liver plays a pivotal role in detoxification but due to its fatty acid content and the lipid soluble characteristics of neurotoxins, lipid based interventions are required to impact toxic burdens. Once the liver has been damaged it can no longer metabolize fats normally. Pools of lipids are then deposited within hepatocytes throughout the liver. Beta oxidation of fatty acids is suppressed impairing detoxification and prostaglandin production. Extensive research with PC has revealed that it protects the liver against damage from alcohol, pharmaceuticals, environmental pollutants, xenobiotics and infection due to viral, bacterial and fungal manifestations (Lieber 1994a, 1994b, 1995, 2001a, 2001b).